mouse skeletal muscle fibroblast myoblast cells (ATCC)
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mouse skeletal muscle fibroblast myoblast cells
Mouse Skeletal Muscle Fibroblast Myoblast Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 8605 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+skeletal+muscle+fibroblast+myoblast+cells/C2C12/pm41237469-161-4-15
Average 99 stars, based on 8605 article reviews
Mouse Skeletal Muscle Fibroblast Myoblast Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 8605 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+skeletal+muscle+fibroblast+myoblast+cells/C2C12/pm41237469-161-4-15
Average 99 stars, based on 8605 article reviews
mouse skeletal muscle fibroblast myoblast cells - by Bioz Stars,
2026-09
99/100 stars
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Related Articles
Cell Culture:Article Title: Integrative multi-omics and single-cell dissection of the immune-fibrotic microenvironment in Duchenne muscular dystrophy reveals therapeutic potential of hesperidin. Article Snippet: Background: Duchenne muscular dystrophy (DMD) is a severe pediatric, X-linked muscle-wasting disorder marked by chronic inflammation, incomplete regeneration, and progressive fibrosis.. The immune–fibrotic microenvironment and its regulatory targets remain insufficiently defined.. We sought to identify hub genes central to DMD pathology, delineate their functional and immune associations, and evaluate the therapeutic potential of hesperidin. Modification:Article Title: Integrative multi-omics and single-cell dissection of the immune-fibrotic microenvironment in Duchenne muscular dystrophy reveals therapeutic potential of hesperidin. Article Snippet: Background: Duchenne muscular dystrophy (DMD) is a severe pediatric, X-linked muscle-wasting disorder marked by chronic inflammation, incomplete regeneration, and progressive fibrosis.. The immune–fibrotic microenvironment and its regulatory targets remain insufficiently defined.. We sought to identify hub genes central to DMD pathology, delineate their functional and immune associations, and evaluate the therapeutic potential of hesperidin. |